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Is non-sustained ventricular tachycardia a key player in non-ischemic cardiomyopathy?
Session:
SESSÃO DE POSTERS 44 - ARRITMIAS VENTRICULARES
Speaker:
Mariana Rodrigues Simões
Congress:
CPC 2025
Topic:
F. Valvular, Myocardial, Pericardial, Pulmonary, Congenital Heart Disease
Theme:
17. Myocardial Disease
Subtheme:
17.2 Myocardial Disease – Epidemiology, Prognosis, Outcome
Session Type:
Cartazes
FP Number:
---
Authors:
Mariana Rodrigues Simões; Luísa Gomes Rocha; Diogo Fernandes; Tatiana Pereira Dos Santos; João Ferreira; Luís Paiva; Lino Gonçalves
Abstract
<p style="text-align:justify"><span style="font-size:12pt"><span style="font-family:"Times New Roman",serif"><strong><span style="font-family:"Arial",sans-serif">Introduction: </span></strong><span style="font-family:"Arial",sans-serif">Patients with non-ischemic cardiomyopathy (NICM) frequently exhibit non-sustained ventricular tachycardia (NSVT) during </span><span style="font-family:"Arial",sans-serif">cardiac implantable electronic device (CIED) </span><span style="font-family:"Arial",sans-serif">follow-up, but studies on its role remain limited and inconclusive. </span></span></span></p> <p style="text-align:justify"><span style="font-size:12pt"><span style="font-family:"Times New Roman",serif"><strong><span style="font-family:"Arial",sans-serif">Methods and purpose:</span></strong><span style="font-family:"Arial",sans-serif"> We performed a single-centre retrospective, observational study reviewing patients with NICM </span><span style="font-family:"Arial",sans-serif">who received CIED between May 2014 - October 2018, to evaluate NSVT occurrence and associated factors, using clinical records and SPSS software for analysis. </span></span></span></p> <p style="text-align:justify"><span style="font-size:12pt"><span style="font-family:"Times New Roman",serif"><strong><span style="font-family:"Arial",sans-serif">Results: </span></strong><span style="font-family:"Arial",sans-serif">A total of 150 patients were included. </span><span style="font-family:"Arial",sans-serif">Sixty-seven percent of patients were men. </span><span style="font-family:"Arial",sans-serif">Seventy-five patients had a CRT (cardiac resynchronization therapy)-defibrillator, 20 patients had a CRT-pacemaker, and 55 patients had an ICD (implantable cardioverter-defibrillator). Eighty-seven percent of patients implanted the device as primary prevention and 13% as secondary prevention. One-hundred patients had dilated cardiomyopathy, 31 hypertrophic cardiomyopathy and 19 other phenotypes. </span><span style="font-family:"Arial",sans-serif">During a follow-up time of 6.64±3.55 years, 103 patients presented at least 1 non-sustained ventricular tachycardia (NSVT) detected by the device. Patients were divided in NSVT and non-NSVT groups. NSVT events were more common in men (n=75 versus (<em>vs</em>) 28, p=0.034), and no differences in age were found between groups (65.58±12.15 <em>vs</em> 61.94±16.59 years, p=0.132). Creatinine levels were higher in the NSVT group: 1.12 (IQR 0.52) <em>vs</em> 0.94 (IQR 0.39) mg/dl, p=0.039.<em> </em>NSVT patients had lower values of left ventricular ejection fraction (LVEF): 30 (IQR 10) <em>vs </em>34 (IQR 28) %, p=0.019. They also presented higher left ventricular end-diastolic diameter (LVEDD) (66.07± 9.89 vs 59.27± 12.53mm, p=0.001) and left ventricular end-systolic diameter (LVESD) (53.97±11.18 <em>vs </em>43.85±14.57mm, p<0.001). No significant association was found between atrial fibrillation and </span><span style="font-family:"Arial",sans-serif">the occurrence of NSVT (p=0.196). There was no association between appropriated shocks (p=0.243), ventricular tachycardia episodes (p=0.092), ventricular fibrillation episodes (p=0.191) or all-cause mortality (p=0.208) and NSVT events. However, 85% of the patients who underwent anti-tachycardia pacing were in the NSVT group (OR=3.76 (CI 1.34-19.57), p=0.008) and all 8 patients who experienced an electric storm belonged to this group. </span></span></span></p> <p style="text-align:justify"><span style="font-size:12pt"><span style="font-family:"Times New Roman",serif"><strong><span style="font-family:"Arial",sans-serif">Conclusion: </span></strong><span style="font-family:"Arial",sans-serif">Male gender, higher creatinine levels, lower values of LVEF, and higher values of </span><span style="font-family:"Arial",sans-serif">LVEDD and LVESD seem to be associated with the presence of NSVT events. However, in our cohort, NSVT presence was not able to predict ventricular arrhythmias in patients with CIED and NICM.</span></span></span></p>
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